The Literature → Phenibut

Phenibut: 15+ high-signal papers with practical takeaways

Mechanism (GABAΒ & α2δ), U.S. poison-center trends, psychosis/delirium, withdrawal, fatalities, and treatment options (baclofen tapers, benzodiazepines, phenobarbital). All items link to PubMed/PMC.

Updated: Oct 18, 2025 Audience: Clinicians & Parents Format: Short summaries + citations

Curated Articles

Mechanism • α2δ + GABAΒ

R-phenibut binds α2δ and shows gabapentin-like activity

Establishes α2δ engagement in addition to GABAΒ—explains gabapentinoid-like toxicity & withdrawal features.

Summary

Animal antinociception with receptor binding confirms α2δ targeting by R-phenibut; helps explain synergy/overlap with gabapentinoids.

Zvejniece L, et al. R-phenibut binds to the α2–δ subunit of voltage-dependent calcium channels and exerts gabapentin-like antinociception. Neurochem Int. 2015;89:41-47. PubMed

Mechanism • Enantiomers

R-phenibut drives GABAΒ effects; S-phenibut largely inactive

Clarifies potency and expected effects by stereoisomer—important for product variability and dosing risk.

Summary

Compares optical isomers across assays; R-phenibut shows GABAΒ activity consistent with clinical effects.

Dambrova M, et al. Comparative pharmacological activity of optical isomers of phenibut. J Pharm Pharmacol. 2008;60(7):795-802. PubMed

Overview • Classic Review

Foundational review of phenibut’s pharmacology & uses

Historic but influential review summarizing GABA-mimetic actions and early clinical experiences.

Summary

Frames Western literature with former-Soviet usage; useful for context when counseling on imported “nootropics.”

Lapin I. Phenibut (β-phenyl-GABA): a tranquilizer and nootropic drug. CNS Drug Rev. 2001;7(4):471-481. PubMed

Epidemiology • Regional Poison Center

Five-year surge in phenibut calls (U.S. region)

CNS/respiratory depression common; some cases required intubation—front-line ED implications.

Summary

Describes exposure routes, symptoms, and outcomes; flags “supplement” source and polysubstance patterns.

McCabe DJ, et al. Phenibut exposures and clinical effects reported to a regional poison center. Am J Emerg Med. 2019;37(10):1937-1940. PubMed

Epidemiology • National (MMWR)

Phenibut exposures reported to U.S. poison centers (2009–2019)

1,320 exposures; agitation, somnolence, coma, and pediatric cases—national scope across all 50 states.

Summary

Characterizes age distribution, clinical effects, and outcomes; establishes baseline for policy and public-health messaging.

Graves JM, et al. Phenibut Exposures Reported to Poison Centers — United States, 2009–2019. MMWR. 2020;69(35):1227-1232. CDC

Clinical • Toxicity & Withdrawal (Series)

Clinical presentations & treatment of phenibut toxicity/withdrawal

Synthesizes intoxication (psychosis, movement disorders, respiratory depression) and withdrawal courses; many severe cases required intubation or ICU-level care.

Summary

Multi-case review outlines typical meds (benzodiazepines, baclofen, phenobarbital, gabapentinoids) and trajectories; emphasizes long, symptom-driven tapers for severe dependence.

Weleff J, Patel R, D’Souza R, et al. Clinical Presentations and Treatment of Phenibut Toxicity and Withdrawal: Case Series and Literature Review. Psychosomatics / Psychiatry. 2023. PMID: 37579098. PubMed

Dependence • Early Case

Phenibut dependence with rapid tolerance and withdrawal

Early English-language case documenting dose escalation, short inter-dose withdrawal, and functional impairment.

Summary

Highlights availability via online “nootropic” vendors; charts classic withdrawal (anxiety, tremor, insomnia, autonomic symptoms) and urges regulatory attention.

Samokhvalov AV, Paton-Gay CL, Balchand K, Rehm J. Phenibut dependence. BMJ Case Rep. 2013;2013:bcr2012008389. PMC

Dependence • U.S. Case + Review

Phenibut dependence and withdrawal (U.S. case)

Confirms GABAB agonist liability with significant withdrawal; discusses practical management with baclofen and benzodiazepines.

Summary

Case with escalating daily doses, anxiety/insomnia on cessation, and gradual stabilization; includes concise literature review useful for bedside counseling.

Ahuja T, Shah R, Siddiqui WJ. Phenibut (β-phenyl-γ-aminobutyric acid) dependence and withdrawal. Prim Care Companion CNS Disord. 2018;20(3):18l02219. PMC

Withdrawal • Protracted/Delirium

Dissociative intoxication and prolonged withdrawal

Severe, protracted withdrawal with delirium; anticipate longer courses and higher-acuity management.

Summary

Describes dissociation, agitation, insomnia, and cognitive changes over weeks; supports need for slow tapering and close follow-up.

Joshi YB, MacKenzie EM, Narasimhan M. Dissociative intoxication and prolonged withdrawal associated with phenibut. Bosn J Basic Med Sci. 2017;17(3):253-256. PMC

Neuropsychiatry • Psychosis

Acute psychosis associated with phenibut ingestion

Illustrates hallucinations/psychosis after phenibut with co-medications; important differential for agitated delirium in the ED.

Summary

Details time course, workup, and supportive/benzodiazepine management; recommends targeted history for imported “supplements.”

Acosta E, Kamboj A, Afzal A, et al. Acute Psychosis Associated with Phenibut Ingestion. Cureus. 2021;13(11):e19335. PMC

Emergency • Overdose

Emergency-department overdose case

Describes delirium, autonomic instability, and supportive treatment; demonstrates unpredictable dosing of gray-market powders.

Summary

Case underscores importance of airway protection and serial neurologic exams; emphasizes unregulated supplement sourcing as a major risk factor.

Nedzlek CD, et al. A Case Report of Phenibut Overdose. Cureus. 2022;14(5):e24795. PMC

Withdrawal • Delirium

Phenibut intoxication and withdrawal delirium

Highlights mixed receptor targets—GABAB, GABAA, gabapentinoid—and integrated management across these systems.

Summary

Provides a conceptual pharmacologic model for severe withdrawal; stresses early recognition and cross-titration strategies.

Martin R, et al. Phenibut Intoxication and Withdrawal Delirium: A Case Report. J Addict Med. 2023;17(6):e420-e423. PMID: 37930202. PubMed

Forensic • Fatality

Phenibut detected in a death investigation

Confirms phenibut’s lethality at high levels and necessity of toxicologic screening in unexplained fatalities.

Arndt C, et al. Phenibut, a GABAB Agonist, Detected in a Fatality. J Anal Toxicol. 2022;46(2):e73-e79. PubMed

Treatment • Outpatient Baclofen Taper

Outpatient withdrawal management using baclofen taper

Demonstrates a structured ambulatory taper strategy for patients refusing or unable to access inpatient detox.

Summary

Gradual baclofen substitution and taper minimized withdrawal; monitoring plan provided. Useful model for rural or outpatient care.

DiFiore E, et al. A Case of Phenibut Withdrawal Management and Outpatient Baclofen Taper. J Addict Med. 2024;XX(X):XX-XX. PMID: 38993282. PubMed

Systematic Review • Withdrawals

Systematic review of phenibut withdrawal cases

Aggregates 15 cases (mean 13.6 g/day); most treated successfully with baclofen, benzodiazepines, or phenobarbital.

Summary

Provides dosing ranges and timeline benchmarks for symptom resolution; suggests cross-taper protocol similar to GABAB agonist withdrawals.

Stewart C, et al. A Systematic Review of Phenibut Withdrawals. Cureus. 2024;16(6):e60338. PMC

Additional Reading & Mechanistic Context

Mechanism • GABAB Ligands

Structure and activation of GABAB receptors

Clarifies why all GABAB agonists—including phenibut and baclofen—share tolerance and withdrawal profiles.

Pin JP, et al. Structural basis of GABAB receptor activation and drug binding. Front Pharmacol. 2020;11:1092. PMC

Narrative • Misuse Review

Phenibut as an emerging drug of misuse

Reviews clinical features and regulatory challenges of phenibut as an unapproved supplement marketed in the U.S.

Shulgina A, et al. Phenibut: An Emerging Drug of Misuse with GABAB Agonist Properties. Psychiatr Clin Cases. 2019. PMID: 30852710. PubMed

Informational only — not medical advice. Phenibut is not FDA-approved in the U.S.; advise patients to disclose use and avoid abrupt discontinuation.